Dr Gunja Jain
About Me
Dr Gunja Jain
MD
Rheumatologist
10 Years Experience
Biography
My Clinic
OPD Hours
| Monday | Morning | 10:00 AM | - | 02:00 PM |
| Evening | 06:00 PM | - | 09:00 PM | |
| Tuesday | Morning | 10:00 AM | - | 02:00 PM |
| Evening | 06:00 PM | - | 09:00 PM | |
| Wednesday | Morning | 10:00 AM | - | 02:00 PM |
| Evening | 06:00 PM | - | 09:00 PM | |
| Thursday | Morning | 10:00 AM | - | 02:00 PM |
| Evening | 06:00 PM | - | 09:00 PM | |
| Friday | Morning | 10:00 AM | - | 02:00 PM |
| Evening | 06:00 PM | - | 09:00 PM | |
| Saturday | Morning | 10:00 AM | - | 02:00 PM |
| Evening | 06:00 PM | - | 09:00 PM | |
| Sunday | Morning | 10:00 AM | - | 02:00 PM |
| Evening | 06:00 PM | - | 09:00 PM |
Treatments
Specialised care and treatments offered
OSTEOARTHRITIS
A degenerative joint disease marked by breakdown of cartilage leading to bone-on-bone contact causing pain and impaired movement. It generally affects weight-bearing joints such as knees or hips but can involve hands too.
- Clinical features : gradual onset stiffness especially after inactivity combined with aching discomfort during activity progressing through time;
- Pathogenesis : wear-and-tear causes cartilage erosion accompanied by bone remodeling;
- Diagnostic evaluation : primarily based on clinical history corroborated with radiographs revealing joint space narrowing plus osteophyte formation;
- Management principles : focus on symptom relief using analgesics/NSAIDs along with physical therapy aimed at strengthening muscles surrounding joints;
While no cure exists currently for osteoarthritis, lifestyle modifications paired with appropriate medical treatment improve quality of life substantially ensuring better mobility preservation.
GOUT
Gout is a metabolic disorder characterized by sudden episodes of intense joint inflammation due to deposition of urate crystals. It most commonly affects the big toe but can involve other joints as well.
- Symptoms : attacks include severe pain often starting at night accompanied by redness, warmth, swelling around affected joints;
- Causes : elevated uric acid levels in blood lead to crystal formation precipitating acute flares;
- Diagnosis : confirmed through clinical presentation supported by analysis of synovial fluid from affected joint demonstrating needle-shaped crystals;
- Treatment : acute attacks managed using NSAIDs or colchicine; long-term control focuses on urate-lowering therapy via medications like allopurinol alongside lifestyle changes reducing purine-rich foods;
Prompt management facilitates rapid relief from episodes while preventing recurrent attacks preserving joint health over time.
PSORIATIC ARTHRITIS
This type of arthritis occurs in some individuals who have psoriasis, characterized by inflamed joints alongside typical skin changes. It is a chronic condition requiring ongoing care.
- Main symptoms: joint pain, swelling often asymmetrical involving fingers or toes with possible nail abnormalities; skin patches may appear scaly and red with silvery scales.
- Causes: believed to be immune-mediated triggered by genetic susceptibility combined with environmental factors such as infections or trauma to skin/joints.
- Diagnosis process: includes clinical evaluation of skin lesions plus laboratory tests excluding other forms of arthritis along with imaging techniques like MRI or ultrasound for early detection of joint involvement.
- Treatment approaches: range from topical treatments addressing psoriasis to systemic DMARDs and biologics that modulate immune response targeting both skin lesions and joint inflammation simultaneously;
A multidisciplinary approach enhances overall symptom control improving patient’s day-to-day functioning substantially.
ANKYLOSING SPONDYLITIS
A form of inflammatory arthritis predominantly affecting the spine and sacroiliac joints. This condition can cause chronic pain and stiffness leading to reduced spinal mobility if unmanaged.
- Symptoms: mainly lower back pain worsening at night or rest with morning stiffness lasting longer than 30 minutes; fatigue is also common.
- Etiology: linked strongly to genetic factors including HLA-B27 gene presence though environment plays a role too.
- Diagnostic tools: physical exam focused on spinal flexibility tests combined with imaging like MRI to detect early inflammation before visible damage appears on X-rays; blood tests may show elevated inflammatory markers.
- Treatment plan: nonsteroidal anti-inflammatory drugs (NSAIDs) are first line; biologic therapies targeting tumor necrosis factor (TNF) have transformed management for resistant cases; physical therapy supports maintaining posture and flexibility.
Early recognition improves outcomes significantly by slowing structural progression through appropriate intervention strategies.
SYSTEMIC LUPUS ERYTHEMATOSUS
This autoimmune condition affects multiple organ systems including skin, joints, kidneys, and the nervous system. It occurs when the body's immune system mistakenly attacks its own tissues.
- Symptoms: vary widely but often include fatigue, joint pain, skin rashes like butterfly-shaped rash on the face, fever, and organ-specific signs depending on severity.
- Causes: genetic predisposition combined with environmental triggers such as sunlight exposure or infections may initiate disease onset.
- Diagnosis: based on clinical features supported by blood tests including antinuclear antibody (ANA) titers and other specific autoantibodies.
- Treatment: involves immunosuppressive medications like corticosteroids and antimalarials to control flare-ups while preventing organ damage.
- Expectations: close follow-up is essential due to variability in disease activity; lifestyle modifications help manage symptoms effectively.
Lupus requires comprehensive care tailored individually to minimize complications over time.
RHEUMATOID ARTHRITIS
Rheumatoid arthritis (RA) is a chronic autoimmune disorder that primarily affects the joints, causing inflammation and pain. It can lead to joint damage and loss of function if not treated appropriately.
- Symptoms: persistent joint pain, swelling, stiffness particularly in the morning or after inactivity, and fatigue.
- Causes: an abnormal immune response targets the synovium (lining of the joints), though exact triggers remain unclear.
- Diagnosis: involves physical examination, blood tests for rheumatoid factor and anti-CCP antibodies, and imaging such as X-rays or ultrasound to assess joint damage.
- Treatment options: include disease-modifying antirheumatic drugs (DMARDs), biologic agents, corticosteroids, and physical therapy to control inflammation and preserve joint function.
- Management: patients often require long-term monitoring with adjustments in medication to reduce symptoms and prevent progression.
Early diagnosis and treatment are crucial for improving quality of life in RA patients by minimizing joint destruction.